@pappabel
PhD researcher in emergency and intensive care medicine at the University of Pécs. Focusing on post-cardiac arrest brain injury and traumatic brain injury.
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Post-cardiac arrest brain injury model: neurobiomarker kinetics following global cerebral hypoperfusion-reperfusion
Post-cardiac arrest brain injury model: neurobiomarker kinetics following global cerebral hypoperfusion-reperfusion
Post-cardiac arrest brain injury (PCABI) is not only caused by the abrupt cessation of cerebral blood flow (CBF), but also by the subsequent cerebral reperfusion following the return of spontaneous circulation (ROSC).1,2 Pulseless ventricular tachycardia (pVT) is one of the common observed arrhythmia during resuscitation.3 Under pVT the effective heart pump function is reduced near to zero,4 the pulse wave velocity and blood pressure is also markedly reduced, thereby establishing a low-/no-flow state.
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